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tion of the gut and other critical organs in preterm newborns remain to be investigated in both pigs and infants.Background Bronchopulmonary dysplasia (BPD) is a common pulmonary complication in preterm infants. Acetate is a metabolite produced by the gut microbiota, and its anti-inflammatory function is well known. The role of acetate in BPD has not been studied. Here, we investigate the effects of acetate on lung inflammation and damage in mice model of BPD. Objective To investigate the role of acetate in the development of BPD. Method
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