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eposition and TGF-β protein expression in myocardial tissues in DCM mice (p0.05). HE staining results showed that PIO notably attenuated myocardial hypertrophy in DCM mice (p0.05). Furthermore, it was discovered that PIO markedly elevated PTEN protein in myocardial tissues of DCM mice and inhibited the phosphorylation of AKT and FAK proteins (p0.05). The protective effect of PIO against cardiac fibrosis in diabetic mice may be related to its regulation on the PTEN/AKT/FAK signaling pathway. Our findings suggest that PI
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