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In our study, we identified a circRNA circFAT1(e2) with an upregulated expression amount in OS tissues. By useful experiments, we discovered that circFAT1(e2) exhaustion dramatically suppressed the proliferation and decreased migration in OS. With regards to process, we discovered that circFAT1(e2) inhibited miR-181b, while miR-181b targeted HK2. By releasing the inhibition of miR-181b on HK2 appearance, leading to attenuated OS development. Mechanistic investigations rec
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