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This was associated with increased glomerular filtration rate (GFR), urine flow and tubular sodium transport rate (T ). Although both total O delivery and T were increased, more O was extracted per transported sodium in rats treated with high-doses of HIF-PHIs, suggesting a reduction in metabolic efficiency. Changes in RVR and GFR were associated with increased nitric oxide (NO) generation and substantially suppressed by pharmacological inhibition of NO synthesis. Our data provide mechanistic insights into dose-dependent effects of shor
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