https://www.selleckchem.com/products/PI-103.html
Rationale Rheumatoid arthritis (RA) is a prototype of inflammatory arthritis in which synovial fibroblasts (SFs) play key roles in cartilage and bone destruction through tumor-like proliferation, migration, invasion and inflammation. This study aimed to research forkhead box protein C1 (FoxC1) and microRNA (miR)-141-3p, which modulate pathological changes in the synovial membrane, to find possible strategies for treating RA. Methods FoxC1, β-catenin and miR-141-3p gene expression in synovial tissues and SFs was quantified by real-time PC
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