https://www.selleckchem.com/pr....oducts/17-AAG(Geldan
Accordingly, knockdown of GRHL2, combined with estrogen treatment, resulted in increased cell migration but no change in proliferation. These results support a model in which GRHL2 binds to selected enhancers and facilitates pS118-ER recruitment to chromatin, which then results in differential activation and repression of genes that control estrogen-regulated ER-positive breast cancer cell migration.Antimalarial resistance threatens global malaria control efforts. The World Health Organization (WHO) recommends routine antim