https://www.selleckchem.com/pr....oducts/Fulvestrant.h
Meanwhile, miR-302c-5p interacted with the signal transducer and activator of transcription 3 (STAT3) and negatively regulated its expression. Furthermore, our data revealed that KT2 restrained the activation of STAT3 by elevating miR-302c-5p, thereby inhibiting Th17 cell differentiation. KT2 alleviates UC by repressing Th17 cell differentiation through the miR-302c-5p/STAT3 axis. KT2 alleviates UC by repressing Th17 cell differentiation through the miR-302c-5p/STAT3 axis.Lung cancer with worldwide distribution, high incidence and l
Aimer
Commentaire
Partagez
