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A total of 103 compounds were characterized in XHCF. XHCF could effectively improve acute colitis caused by DSS. More to the point, XHCF substantially decreased M1 macrophage markers (CD11c, IL-6 and IL-1β) whereas increased M2 macrophage markers (CD206) in colitis mice, suggesting it could manage macrophage polarization. Moreover, the levels of HK2 and lactic acid in colon tissues had been notably paid off by XHCF, indicating that XHCF could inhibit glycolysis. Additionally down-
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