https://www.selleckchem.com/pr....oducts/Etopophos.htm
This demonstrates that COL7A1 editing based on precise end joining-mediated DNA repair is an efficient strategy to revert the disease-associated nature of DEB, regardless of the mutational inheritance. We have previously shown that endocannabinoids promote sebaceous lipogenesis, and sebocytes are involved in the metabolism of the "endocannabinoid-like" substance oleoylethanolamide (OEA). OEA is an endogenous activator of GPR119, a recently de-orphanized receptor, which is currently being investigated as a promising anti-diabetic drug
Beğen
Yorum Yap
Paylaş
