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Besides, we isolated and cultured IL-23R+ individual tumor cells through the post-operation tumor sample of three LC patients, and discovered that rhIL-23 could phosphorylate STAT3 (pSTAT3, residue Y705), which resulted in cancer tumors cellular proliferation and cisplatin opposition. These results suggest that IL-23R had been a Hallmark of cancer immunoediting process, and focusing on IL-23 is highly recommended as a therapeutic selec
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