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discordant. This study should be extended for larger cohort of patients. Uncontrolled type 1 diabetes mellitus (T1D) impairs reproductive potential of males. Insulin treatment restores metabolic parameters but it is unclear how it protects male reproductive health. Herein, we hypothesized that insulin treatment to T1D rats protects testicular physiology by mediating mechanisms associated with apoptosis and cell cycle. Mature male Wistar rats (n=24) were divided into 3 groups control, T1D-induced (received 40mgkg streptozotocin) and insu
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