https://www.selleckchem.com/products/U0126.html
Aim This study aimed to explore the genetic and epigenetic similarities between chronic obstructive pulmonary disease (COPD) and lung adenocarcinoma (LUAD). Materials methods We mainly used Weighted correlation network analysis, protein-protein interaction network and pivot analysis to identify hub modules, bridge regulators, bridge genes and hub-driving genes in both diseases and carried out verifying using external datasets. Results We identified eight bridge regulators, 19 key molecules in the COPD model and ten key molecules in
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