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To explore this hypothesis, we constructed two M. bovis 'knock-in' strains containing the M. tuberculosis pknH gene. Evaluation of their virulence phenotype in mice revealed a reduced virulence of both M. bovis knock-in strains compared to the wild type, suggesting that PknH plays an important role in the differential virulence phenotype of M. bovis vs M. tuberculosis.[This corrects the article DOI 10.1371/journal.ppat.1008477.].Hodgkin lymphoma (HL) survivors are at increased risk of developing second primary esophageal squamous cell ca
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