https://www.selleckchem.com/products/pds-0330.html
Hyperandrogenism activated the NF-κB pathway. After using the MIF antibody, this effect was reversed. This finding suggested that hyperandrogenism activated the NF-κB pathway through MIF. In short, increased MIF levels activated the NF-κB pathway in ovaries, leading to inflammation and the increase in the levels of relevant inflammatory indicators, which might be one of the important factors in the pathogenesis of PCOS.Although the beneficial effects of metformin (MET) and genistein in ameliorating inflammation have been elucidated, th
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