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Electrophysiological recordings were collected at each visit from both the cortical and subcortical implants. RESULTS Recordings collected from the CM thalamic nucleus revealed a low-frequency power (3-10 Hz) increase that was time-locked to the onset of involuntary tics but was not present during voluntary movements. Cortical recordings revealed beta power decrease in M1 that was present during tics and voluntary movements. CONCLUSION We conclude that a human physiological signal was detected from the CM thalamus that differentiated tic
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