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Outcomes showed that intra-CA1 administration of SCH-23390 or sulpiride could stop the intra-LH carbachol-induced antinociception. This result ended up being a tad bit more prominent after preventing the D2-like dopamine receptor when you look at the CA1. These findings disclosed that D1- and D2-like dopamine receptors in the CA1 perform an important role in antinociceptive answers caused by chemical stimulation
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