https://www.selleckchem.com/products/sq22536.html
IL-36RN overexpression inhibited the proliferation, migration, invasion, and colony formation in RCC cells via suppressing β-catenin. In the xenograft mouse model, delivering recombinant IL-36RN inhibited tumor formation in vivo. CONCLUSION We demonstrated here that IL-36RN could suppress the development of RCC, which might be developed as a novel independent prognostic factor and therapeutic target. V.INTRODUCTION This multicentre, retrospective study aimed to establish correlation between estimated tumour volume doubling times (TVDT)
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