https://www.selleckchem.com/products/prgl493.html
, EEF1A2 promoted the proliferation, migration, and invasion of OS cells; , EEF1A2 promoted the growth of OS tumors. The mechanism study showed that EEF1A2 can promote protein kinase B (Akt) and mammalian target of rapamycin (mTOR) phosphorylation, thereby activating the Akt/mTOR signaling pathway in OS. There is high expression of EEF1A2 in OS, which can promote the proliferation, migration, and invasion of OS cells and the growth of OS tumors in vivo via activation of the Akt/mTOR signaling pathway. There is high expression of EEF1A2
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