https://www.selleckchem.com/pr....oducts/incb28060.htm
Ischemic preconditioning (IP) reduces brain damage after subsequent ischemic strokes by activating endogenous protective mechanisms in rodents. Transient ischemic attack (TIA) induces tolerance in the human brain after ischemic strokes; defining mechanisms of IP effects may provide therapeutic targets to improve recovery of patients with ischemic strokes. Iron transported across the blood-brain barrier (BB is required for brain functions, including myelination, and its levels should be finely regulated to avoid harmful effects. This
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