https://www.selleckchem.com/pr....oducts/enpp-1-in-1.h
Glycolysis controls mTORC1 signaling and protein synthesis. In skeletal muscle, glucose metabolism increases with both exercise/contraction intensity and volume, and therefore, high-intensity muscle contraction (HiMC) such as resistance exercise facilitates glycolysis including glucose uptake and glycogen breakdown. However, it is unknown whether glycolysis regulates HiMC-induced mTORC1 activation and increase in protein synthesis. To determine whether glycolysis regulates basal and HiMC-induced mTORC1 signaling and protein synthesi
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