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Cardiac exosomes (Exos) have already been reported concerning fibrosis in ischemic cardiomyopathy. Hence, we investigated whether Exos secreted from the familial DCM cardiomyocytes could market fibrogenesis. Using human iPSCs differentiated cardiomyocytes we isolated Exos of angiotensin II stimulation conditioned media from either DCM or control (CTL) cardiomyocytes. Of great interest, cultured cardiac fibroblasts had increased fibrogenesis after contact with DCM-Exos rather tha
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