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DEX also considerably promoted the release of ROS. However, DEX (≥100 μM) downregulated the protein expression of Bcl-2, decreased the mitochondrial membrane potential (MTP), and reduced ATP synthesis. High concentrations of dexmedetomidine produced toxic effects on neurons and caused neuronal apoptosis. High concentrations of dexmedetomidine produced toxic effects on neurons and caused neuronal apoptosis.[This retracts the article DOI 10.2147/DDDT.S249557.].Monoclonal antibodies (mAbs) are part of the standard of care for the treatment
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