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Additionally, we found that HP played the role in accelerating VBNC formation in the process of HPCD treatment. Transcriptomic analysis revealed 85, 263 and 529 differentially expressed genes (DEGs) for HP-, pHex3- and HPCD-treated cells compared with untreated ones. 59 DEGs shared by pHex3 and HPCD treatment might be responsible for VBNC induction, and they were mainly involved in cellular transport and localization.For assessing the carotenoid bioaccessibility during in vitro digestion, obtaining the micelle fraction that can diffuse t
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