https://cp640186inhibitor.com/....type-4-hiatal-hernia
Linc-UBC1 ended up being primarily distributed in the nucleus. Silence of linc-UBC1 attenuated proliferative and migratory capabilities of HO8910 and HEY cells. RIP assay confirmed that linc-UBC1 could restrict the transcription of p53. Knockdown of p53 could partially reverse the regulatory effects of linc-UBC1 on regulating the development of OC. CONCLUSIONS Linc-UBC1 is upregulated in OC tissues and cells. It stimulates the proliferation and metastasis of OC by downregulating p53 level, thus exerti
Me gusta
Comentario
Compartir
