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More over, optimization associated with CD32+CD4+ T cellular purification protocol reveals prominent enrichment for HIV DNA (mean, 292-fold) during these cells. Nevertheless, no enrichment for HIV RNA is observed in CD32+CD4+ cells, yielding significantly decreased HIV RNA/DNA ratios. Furthermore, HIV proviruses in CD32+CD4+ cells can be reactivated ex vivo to produce virus, strongly recommending why these cells support HIV transcriptional latency. Our results underscore the significanc
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