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early life remains unknown. Here we document persistent deficits in spinal inhibitory circuits involving dynorphin-lineage (DYN) interneurons previously implicated in gating mechanical pain and itch. Notably, neonatal injury reduced the strength of DYN inhibitory synapses onto mature lamina I spinoparabrachial neurons, a major output of the spinal nociceptive network, which could contribute to the priming of pain pathways by early tissue damage. Copyright © 2020 Brewer et al.Despite the success of reperfusion therapy in significantly re
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