https://www.selleckchem.com/products/wz4003.html
In addition, aliskiren could alleviate neuronal inflammatory damage and apoptosis arise from Aβ 1-42accumulation. Following confirming the high expression level of the predicted target PDE4B in damaged cells, it was found that PDE4B overexpression can reverse the influence of aliskiren on cell viability, inflammatory damage and apoptosis. In conclusion, aliskiren upregulates cell viability, reduces inflammatory damage and apoptosis induced by Aβ accumulation in AD via inhibiting PDE4B. These findings have expanded the scope of future app
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