https://www.selleckchem.com/pr....oducts/pf-00835231.h
Furthermore, the ( ) was identified as a direct target of miR-519d-5p and could indirectly upregulate expression by sponging miR-519d-5p. Moreover, the cancer-inhibiting activities of knockdown in NSCLC cells were partially offset by miR-519d-5p inhibition. increases expression by sequestering miR-519d-5p, thereby aggravating the malignant progression of NSCLC. The LINC01426/miR-519d-5p/ETS1 competing endogenous RNA pathway may provide a target for designing therapeutic agents for NSCLC treatment. LINC01426 increases ETS1 expression
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