https://www.selleckchem.com/products/wh-4-023.html
Vitamin D deficiency and altered body composition are common in Alzheimer's disease (AD). Memantine with vitamin D supplementation can protect cortical axons against amyloid-β exposure and glutamate toxicity. To study the effects of vitamin D deprivation and subsequent treatment with memantine and vitamin D enrichment on whole-body composition using a mouse model of AD. Male APPswe/PS1dE9 mice were divided into four groups at 2.5 months of age the control group (n = 14) was fed a standard diet throughout; the remaining mice were starte
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