https://www.selleckchem.com/pr....oducts/hc-030031.htm
FXYD1 (an auxiliary subunit, which modulates Na,K-ATPase activity) abundance and its association with the α2 Na,K-ATPase were decreased in both mouse models of myodystrophy, presumably as response to impaired functioning of the enzyme. Soleus muscles from mdx and Bla/J mice demonstrated difference in the pattern of the α2 Na,K-ATPase and cholesterol abnormalities compared to diaphragm muscles. Our findings indicate that these distinct mouse models of myodystrophy are characterized by α2 Na,K-ATPase and membrane cholesterol impairments
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