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Our results tend to suggest that iron inactivates Parkin in SH-SY5Y cells and thereby inhibits the proteasomal degradation of α-synuclein, and the accumulated α-synuclein causes mitochondrial dysfunction and cell death. These results have implications in the pathogenesis of sporadic Parkinson's disease and also familial type with Parkin mutations. Copyright 2020 The Author(s).Exploring high-efficiency, stable, and cost-effective electrocatalysts for electrochemical activities is greatly desirable and challenging. Herein, a newly designed h
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