https://piperlonguminechemical.....com/redox-post-tran
Linc-UBC1 had been primarily distributed when you look at the nucleus. Silence of linc-UBC1 attenuated proliferative and migratory abilities of HO8910 and HEY cells. RIP assay verified that linc-UBC1 could restrict the transcription of p53. Knockdown of p53 could partly reverse the regulating effects of linc-UBC1 on controlling the development of OC. CONCLUSIONS Linc-UBC1 is upregulated in OC areas and cells. It promotes the expansion and metastasis of O
Gefällt mir
Kommentar
Teilen
