https://www.selleckchem.com/pr....oducts/Cryptotanshin
Third, Ncr1-mediated deletion of Raptor, rather than Rictor, moderately affected NK cell terminal differentiation. In terms of mechanism, mTORC1 mainly promotes the expression of NK cell-specific transcription factor E4 promoter-binding protein 4 (E4BP4), while both mTORC1 and mTORC2 can enhance the expression of T-bet. Therefore, mTORC1 and mTORC2 subtly coordinate NK cell development by differentially inducing E4BP4 and T-bet.The DNA-PK maintains cell survival when DNA damage occurs. In addition, aberrant activation of the DN
Respect!
Kommentar
Delen
