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In animal models, the co-injection of cancer cells and TSG6-reprogrammed fibroblasts led to a significant increase in tumor metastasis. Our findings indicated that TSG-6 overexpression in CRC cells could promote cancer metastasis in both an autocrine and paracrine manner. Therefore, targeting TSG-6 might be a potential therapeutic strategy for the treatment of metastatic CRC.[This corrects the article DOI 10.7150/ijbs.57164.].Background Mannan-binding lectin (MBL), a soluble pattern recognition molecule in the innate immune system, is repo
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