https://www.selleckchem.com/pr....oducts/PIK-75-Hydroc
GLU reduced serum cortisol concentration at 4 h after LPS stress and downregulated the mRNA expression of intestinal corticotropin-releasing factor signal (corticotrophin-releasing factor [CRF], CRF receptor 1 [CRFR1], glucocorticoid receptor, tryptase, nerve growth factor, tyrosine kinase receptor A), and prevented mast cell activation. GLU upregulated the mRNA expression of intestinal transforming growth factor β. These findings indicate that GLU attenuates LPS-induced intestinal mucosal barrier injury, which is associate
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