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g IL1, TNFα, IFNα and IL6). Uptake of urinary exosomes from responders by mesangial cells had been exceptional when compared with that from non-responders (90% vs. 50%, p less then 0.0001). HIF1A had been identified as a possible typical target, and reasonable necessary protein levels had been found in non-responder renal biopsies. HIF1A inhibition decreased mesangial proliferation and IL-8, CCL2, CCL3, and CXCL1 mesangial cell manufacturing and IL-6/VCAM-1 in endothelial cell
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