https://www.selleckchem.com/products/cpi-203.html
In contrast to fibrosis, transitional AECs may retain the capacity for physiologic AEC1 regeneration with restoration of normal alveolar architecture and function. Novel therapies to promote AEC1 differentiation from transitional cells may accelerate barrier restitution and clinical recovery in ARDS.Acute lung immunity to inhaled pathogens elicits defensive pneumonitis that may convert to the Acute Respiratory Distress Syndrome (ARDS), causing high mortality. Mechanisms underlying the conversion are not understood, but are of intense in
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