https://www.selleckchem.com/
5±11.9 vs 34.9%±13.9%; p=0.02) and tracked more closely with abnormal wall motion (r =0.72 vs 0.55; p0.0001). Kinetic modelling showed reduced myocardial manganese influx between remote, peri-infarct and infarct regions, enabling absolute discrimination of infarcted myocardium. After 3 months, manganese uptake increased in peri-infarct regions (16.5±3.5 vs 22.8±3.5 mL/100 g/min, p0.0001), but not the remote (23.3±2.8 vs 23.0±3.2 mL/100 g/min, p=0.8) or infarcted (11.5±3.7 vs 14.0±1.2 mL/100 g/min, p0.1) myocardium. Through visualisation of intra
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