https://www.selleckchem.com/pr....oducts/eidd-2801.htm
AR inhibition increased the expressions of PPAR-α and PPAR-γ in both normal liver and fatty liver groups after major hepatectomy and IR injury. In addition, the knockout of AR promoted the expressions of SDHB, AMPK, SIRT1, and PGC1-α and PPAR. Conclusions The knockout of AR promoted the regeneration of normal and fatty livers through regulating energy metabolism. AR may be a new potential therapeutic target to accelerate liver regeneration after surgery. Copyright © 2020 Chang Xian Li et al.Although the mitochondrial antiviral signali
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