https://www.selleckchem.com/products/bi-2852.html
Based on recent findings, we will discuss how the compartments in living neurons are eliminated for the neuronal circuit plasticity.The defining characteristic of prion diseases is conversion of a cellular prion protein (PrPC) to an abnormal prion protein (PrPSc). The exogenous attachment of PrPSc to the surface of a target cell is critical for infection. However, the initial interaction of PrPSc with the cell surface is poorly characterized. In the current study, we specifically focused on the association of PrPSc with cells during the
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