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Results the consequence of MTT assay demonstrated that UA could restrict MCF-7 mobile growth with IC50 values of 20 μM. Microarray and CMAP analysis, validated by Western blot, suggested that UA considerably modulated IKK/NF-κB, RAF/ERK paths, and down-regulated the phosphorylation level of PLK1 in MCF-7 cells. Conclusion Our data indicated that the anti-tumor effects of UA are caused by the inhibited RAF/ERK path and IKK/NF-κB path. It could also be explained by t
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