https://dub-receptor.com/index.....php/health-related-
Results the consequence of MTT assay demonstrated that UA could restrict MCF-7 mobile growth with IC50 values of 20 μM. Microarray and CMAP analysis, validated by Western blot, suggested that UA considerably modulated IKK/NF-κB, RAF/ERK paths, and down-regulated the phosphorylation level of PLK1 in MCF-7 cells. Conclusion Our data indicated that the anti-tumor effects of UA are caused by the inhibited RAF/ERK path and IKK/NF-κB path. It could also be explained by t
Aimer
Commentaire
Partagez
